RUHS, Jaipur, M.Sc Nursing Entrance Exam-2015
Medical Surgical Nursing
Easy

Clinical manifestation of patient with gastrointestinal bleeding are?

Appeared in: RUHS, Jaipur, M.Sc Nursing Entrance Exam-2015

Explanation

  • This option correctly identifies the classic triad of acute upper GI bleeding: hematemesis, tachycardia, and hypotension.
  • Acute bright red hematemesis signifies active, ongoing bleeding from a source above the ligament of Treitz (e.g., peptic ulcer, esophageal varices).
  • A rapid pulse (tachycardia) is the body's primary compensatory mechanism to maintain cardiac output in response to hypovolemia (blood loss).
  • A drop in blood pressure (hypotension) occurs when blood loss is significant (typically more than 15-20% of total blood volume) and compensatory mechanisms begin to fail.

Why Other Options Were Wrong

  • Option A: While collapse can occur, a 'low feeble pulse' (bradycardia) is not the typical initial response to acute blood loss. Tachycardia (a rapid pulse) is the expected compensatory sign. Bradycardia is a very late and ominous sign indicating decompensation and impending cardiac arrest.
  • Option B: This option is partially correct as it includes low blood pressure and hematemesis. However, it is incomplete because it omits the crucial compensatory sign of a rapid pulse (tachycardia), which is one of the earliest indicators of significant blood loss.
  • Option D: This combination of symptoms is more indicative of chronic or mild blood loss leading to anemia (causing fatigue). The body may be compensating with a rapid pulse, but the normal blood pressure suggests the blood loss is not severe enough to cause hemodynamic instability.

Related Visual

Visual explanation — Related Visual
Clinical Relevance
  • Nursing practice connection: Use the key finding related to The concept tested is the recognition of the classic signs and symptoms of acute gastrointestinal (GI) bleeding, which involves understanding the body's physiological response to significant blood loss to guide bedside assessment, documentation, and the next nursing action.
  • Recognizing the signs of acute GI bleeding is a critical nursing skill. Prompt identification of tachycardia and hypotension allows for rapid intervention to prevent progression to irreversible shock.
  • The nurse's first actions for a patient with suspected acute GI bleed are to ensure airway, breathing, and circulation (ABCs), establish large-bore IV access for resuscitation, and notify the physician immediately.
  • What if? If the patient presented with fatigue and dark, tarry stools (melena) over several weeks with stable vital signs, the priority would shift from immediate resuscitation to diagnostic workup for a chronic or slow upper GI bleed, likely on an outpatient or less urgent basis.
How to Approach the Question
  • First, analyze the question stem to identify the core concept: 'clinical manifestation of gastrointestinal bleeding'.
  • Recognize that GI bleeding can be acute or chronic, and the signs will differ. The options describe acute scenarios.
  • Evaluate each option by considering the pathophysiology of acute blood loss (hemorrhage). The body's response involves a sequence: blood loss -> decreased volume -> compensatory tachycardia -> decompensation with hypotension.
  • Look for the option that provides the most complete and accurate picture of this physiological cascade.
  • Option A is incorrect because the initial pulse response is rapid, not low.
  • Option B is incomplete as it misses the key sign of tachycardia.
Concept Tested & Keywords
  • Concept Tested: The concept tested is the recognition of the classic signs and symptoms of acute gastrointestinal (GI) bleeding, which involves understanding the body's physiological response to significant blood loss.
  • Stem keywords: Clinical manifestation, gastrointestinal bleeding
  • Lead-in keywords: BEST, MOST RELEVANT CLUE

Question ID

QocdiN2Fd8JeQZYiSWvsHO

Reference Book

E6 Medicine Harrison 22e Part 1 pp. 360-362, 357-359

E6 Medicine Macleod Clinical Examination 15e p. 120-122

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